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Every day we are exposed to foods that could cause us food intolerance like lactose intolerance to drugs that can give us side effects that are due to our body reacting negatively to it. Even the simple pleasure of spending time in the backyard expose you to other possible allergens like grass and flowers.Understanding your allergy symptoms and treating the underlying causes are two very important parts of finding an effective solution to improving your health and wellness. There are many ways that people treat allergy symptoms. The band-aid approach of treating the allergic reaction quickly is pretty popular, yet often these treatments are short-lived. With patience and research, one can learn to manage these otherwise debilitating allergies.
Common allergy symptoms are seen throughout the world by both Allergists and Family Practitioners. Doctors tend to categorize the symptoms as mild, moderate or severe. The most common fall into the mild and moderate category and include:
* Chronic Sinusitis
* Watery Eyes that can become Red and Itchy
* Rash
* Congestion
* Itchiness
* Difficulty breathing

Chronic sinusitis is possibly the most frequent sign of an allergy. This usually causes sinus headaches as the nasal passages are continually clogged and sometimes draining as well. Watery eyes are another very common allergy symptom which can cause painful irritation. This causes scratching which can make matters worse particularly in children who usually show no restraint. In order to treat these symptoms, there are a variety of medications available to help stop this reaction and inflammation process. Both prescription and over-the counter medicines are available. Eye drops like Visine are often used as well to take out redness and irritation from the eyes. Natural approaches popular for treating so many things nowadays are also an option when it comes to treating allergy symptoms as well. An example of this is the herb milk thistle which cleanses the liver and helps it to function properly which helps those with food intolerance.

Severe reaction otherwise known as anaphylaxis is more rare but is life-threatening. It may begin with a common symptom like itching of the eyes or face but will suddenly progress to include:
* Swelling that effects breathing and swallowing
* Abdominal pain
* Cramps
* Vomiting
* Diarrhea
* Dizziness (possible mental confusion)
If you think you are experiencing anaphylaxis seek emergency help immediately.

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As discussed previously, IBS is believed to be due to the abnormal function (dysfunction) of the muscles of the organs of the gastrointestinal tract or the nerves controlling the organs. The nervous control of the gastrointestinal tract, however, is complex. A system of nerves runs the entire length of the gastrointestinal tract from the esophagus to the anus in the muscular walls of the organs. These nerves communicate with other nerves that travel to and from the spinal cord. Nerves within the spinal cord, in turn, travel to and from the brain. (The gastrointestinal tract is exceeded in the numbers of nerves it contains only by the spinal cord and brain.) Thus, the abnormal function of the nervous system in IBS may occur in a gastrointestinal muscular organ, the spinal cord, or the brain.
The nervous system that controls the gastrointestinal organs, as with most other organs, contains both sensory and motor nerves. The sensory nerves continuously sense what is happening within the organ and relay this information to nerves in the organ's wall. From there, information can be relayed to the spinal cord and brain. The information is received and processed in the organ's wall, the spinal cord, or the brain. Then, based on this sensory input and the way the input is processed, commands (responses) are sent to the organ over the motor nerves. Two of the most common motor responses in the intestine are contraction or relaxation of the muscle of the organ and secretion of fluid and/or mucus into the organ.
As already mentioned, abnormal function of the nerves of the gastrointestinal organs, at least theoretically, might occur in the organ, spinal cord, or brain. Moreover, the abnormalities might occur in the sensory nerves, the motor nerves, or at processing centers in the intestine, spinal cord, or brain. Some researchers argue that the cause of functional diseases is abnormalities in the function of the sensory nerves. For example, normal activities, such as stretching of the small intestine by food, may give rise to abnormal sensory signals that are sent to the spinal cord and brain, where they are perceived as pain.
Other researchers argue that the cause of functional diseases is abnormalities in the function of the motor nerves. For example, abnormal commands through the motor nerves might produce a painful spasm (contraction) of the muscles. Still others argue that abnormally functioning processing centers are responsible for functional diseases because they misinterpret normal sensations or send abnormal commands to the organ. In fact, some functional diseases may be due to sensory dysfunction, motor dysfunction, or both sensory and motor dysfunction. Still others may be due to abnormalities within the processing centers One area that is receiving a great deal of scientific attention is the potential role of gas produced by intestinal bacteria in patients with IBS. Studies have demonstrated that patients with IBS produce larger amounts of gas than individuals without IBS, and the gas may be retained longer in the small intestine. Among patients with IBS, abdominal size increases over the day, reaching a maximum in the evening and returning to baseline by the following morning. In individuals without IBS, there is no increase in abdominal size during the day.
There has been a great deal of controversy over the role that poor digestion and/or absorption of dietary sugars may play in aggravating the symptoms of IBS. Poor digestion of lactose, the sugar in milk, is very common as is poor absorption of fructose, a sweetener found in many processed foods. Poor digestion or absorption of these sugars could aggravate the symptoms of IBS since unabsorbed sugars often cause increased formation of gas.Although these abnormalities in production and transport of gas could give rise to some of the symptoms of IBS, much more work will need to be done before the role of intestinal gas in IBS is clear.

Dietary fat in healthy individuals causes food as well as gas to move more slowly through the stomach and small intestine. Some patients with IBS may even respond to dietary fat in an exaggerated fashion with greater slowing. Thus, dietary fat could--and probably does--aggravate the symptoms of IBS.  but also because in truth Digestrol™ simply does what YOU need it to do!!!

The future of IBS depends on our increasing knowledge of the processes (mechanisms) that cause IBS. Acquiring this knowledge, in turn, depends on research funding. Because of the difficulties in conducting research in IBS, this knowledge will not come quickly. Until we have an understanding of the mechanisms of IBS, newer treatments will be based on our developing understanding of the normal control of gastrointestinal function, which is proceeding more rapidly. Specifically, there is intense interest in intestinal neurotransmitters, which are chemicals that the nerves of the intestine use to communicate with each other. The interactions of these neurotransmitters are responsible for adjusting (modulating) the functions of the intestines, such as contraction of muscles and secretion of fluid and mucus.5-hydroxytriptamine (5-HT or serotonin) is a neurotransmitter that stimulates several different receptors on nerves in the intestine, including one called the 5-HT4 receptor. Stimulation of these receptors by 5-HT increases muscle contractions in the colon. An example of an experimental drug that affects intestinal neurotransmission is tegaserod. This drug mimics the effect of 5-HT on the 5-HT4 receptor. Thus, because it increases intestinal muscle contractions, tegaserod is being tested for effectiveness in treating constipation-predominant IBS as well as constipation in general.
Another drug that affects neurotransmission is ondansetron. This drug blocks a different receptor, the 5-HT3 receptor, and thereby reduces colonic contractions. Thus, ondansetron (Zofran) has been effective in treating diarrhea-predominant IBS in initial studies. There are drugs that stimulate another receptor, the 5-HT1 receptor. Examples of this type of drug are sumatriptan (Imitrex) and buspirone. These drugs are believed to reduce the responsiveness (sensitivity) of the sensory nerves to what's happening in the intestine. The 5-HT1 receptor stimulators, however, have not yet been tested for effectiveness in IBS. Finally, in preliminary studies, fedotozine has been shown to improve functional gastrointestinal symptoms. The mechanism of action of fedotozine is not known, but it also may act by reducing the sensitivity of the sensory nerves.Finally, there is the issue of a relationship between IBS and intestinal bacteria. Over the next few years, much information will accumulate on this potential relationship.

IBS At A Glance
* IBS is a functional disease, that is, a disease in which the intestine (bowel) functions abnormally.
* Theories of the cause of IBS include abnormal input from intestinal sensory nerves, abnormal processing of input from the sensory nerves, and abnormal stimulation of the intestines by the motor nerves.
* The primary symptoms of IBS are constipation, diarrhea, and abdominal pain. Secondary symptoms include abnormal passage of stool, abnormal form of stool, increased amounts of mucus in the stool, and a subjective feeling of abdominal distention (bloating).
* IBS is diagnosed on the basis of typical symptoms (Rome II Criteria) and the absence of other intestinal and non-intestinal diseases that might give rise to the symptoms. Testing in IBS is directed primarily at excluding the presence of other intestinal diseases and non-intestinal diseases.
* Treatment of IBS consists primarily of medications to control constipation, diarrhea, and abdominal pain. Anti-depressant medication and psychological treatments also may be used. It is not clear if dietary alterations have much effect on the symptoms of IBS except for increases in dietary fiber, which may improve constipation.
* Although it has been hypothesized that IBS may be caused by intestinal bacteria, specifically by small intestinal bacterial overgrowth, there is little rigorous scientific support for the hypothesis.
* Future advances in the treatment of IBS depend on a clearer understanding of its cause(s).

The complications of functional diseases of the gastrointestinal tract are relatively limited. Since symptoms are most often provoked by eating, patients who alter their diets and reduce their intake of calories may lose weight. Fortunately, loss of weight is unusual in functional diseases, and it should suggest the presence of a non-functional disease. Symptoms that awaken patients from sleep also are more likely to be due to non-functional than functional diseases.Most commonly, functional diseases interfere with the patients' comfort and their daily activities. For example, patients who suffer from morning diarrhea may not leave the house until the diarrhea stops. If the diarrhea is constant, they may go only to places where they know that a toilet is readily available. Patients who develop pain after eating may skip lunch. Very commonly, patients associate symptoms with specific foods, such as milk, fat, vegetables, etc. Whether or not these associations are real, these patients will restrict their diets accordingly. Milk is the food that is most commonly eliminated, often unnecessarily and to the detriment of adequate calcium intake. The interference with daily activities also can lead to problems with interpersonal relationships, especially with spouses. However, most patients with functional disease tend to just live with their symptoms and infrequently visit physicians for diagnosis and treatment.

How is IBS diagnosed?The Rome II Criteria
The symptoms of IBS are varied and inconsistent among patients. Moreover, there are no characteristically abnormal tests that can be used to diagnose IBS. All of this has made it difficult to define IBS and identify patients, especially for research studies. In 1999, a group of international investigators met in Rome for a second time (Rome II). There, they developed a set of criteria for symptoms to be used for diagnosing IBS.The Rome II Criteria state that in order to be diagnosed with IBS, a patient should have suffered abdominal pain or discomfort for 12 weeks or more (not necessarily consecutive weeks) in the previous 12 months. The pain or discomfort should have two out of the three following features:
* Relief with defecation
* Onset associated with a change in the frequency of stool
* Onset associated with a change in the form of stool
Other symptoms that are not essential, but support a diagnosis of IBS, are: (1) abnormal frequency of stools (more than 3/day or less than 3/week); (2) abnormal stool form (lumpy and hard, or loose and watery); (3) abnormal stool passage (straining, urgency, or feeling of incomplete evacuation); (4) passage of mucus; and (5) bloating (feeling of abdominal distention, or enlargement).
The Rome II criteria are rather specific for a diagnosis of IBS. In essence, they require the presence of prolonged abdominal pain or discomfort that is in some way related to an alteration in the pattern of bowel movements. Symptoms of dyspepsia (nausea or abdominal discomfort following meals), abdominal distention, and increased flatus (passing gas, or flatulence) alone do not fall within this definition. Nevertheless, many patients have these symptoms along with the symptoms of IBS. It is not clear if these patients have one problem (IBS) or more than one problem.

The Solution is Digestrol™
There are many questions as to what may be promoting all these cases of IBS, i.e. lifestyle, environment, drug reactions, diet, stress, genetics... the list goes on. But Digestrol™ is the ONLY solution that addresses the root cause of the actual problem at a cellular level. The success of Digestrol™ is its ability to soothe all the symptoms simultaneously, not just one or two.

The primary purpose of the gastrointestinal tract is to digest (break down) and absorb (take into the blood stream) food. In order to fulfill this purpose, food must be ground, mixed, and transported through the intestines, where it is digested and absorbed. In addition, undigested and unabsorbed portions of the food must be eliminated from the body.

In functional diseases of the gastrointestinal tract, the grinding, mixing, digestion, and absorption functions are disturbed to only a minor degree. These functions are essentially maintained, perhaps because of a built-in over-capacity of the gastrointestinal tract to perform these functions. The most commonly affected function in these diseases is transportation. In the stomach and small intestine, the symptoms of slowed transportation are nausea, vomiting, abdominal bloating (the sensation of abdominal fullness), and abdominal distention (enlargement). The symptom of rapid transportation usually is diarrhea. The interpretation of symptoms, however, may be more complicated than this. For example, let's say that a person has abnormally rapid emptying of the stomach. The sensing of this rapid emptying by the intestinal sensory nerves normally brings about a motor nerve response to slow emptying of the stomach and transportation through the small intestine. Thus, rapid emptying of the stomach may give rise to symptoms of slowed transportation.

In the colon, abnormally slowed or rapid transportation results in constipation or diarrhea, respectively. In addition, there may be increased amounts of mucus coating the stool or a sense of incomplete evacuation after a bowel movement.

As discussed previously, normal sensations may be abnormally processed and perceived. Such an abnormality could result in abdominal bloating and pain. Abnormally processed sensations from the gastrointestinal organs also might lead to motor responses that cause symptoms of slowed or rapid transportation.

Slowed transportation of digesting food through the small intestine may be complicated, for example, by bacterial overgrowth. In bacterial overgrowth, gas-producing bacteria that are normally restricted to the colon move up into the small intestine. There, they are exposed to greater amounts of undigested food than in the colon, which they turn into gas. This formation of gas can aggravate bloating and/or abdominal distention and result in increased amounts of flatus (passing gas, or flatulence) and diarrhea.

The gastrointestinal tract has only a few ways of responding to diseases. Therefore, the symptoms often are similar regardless of whether the diseases are functional or non-functional. Thus, the symptoms of both functional and non-functional gastrointestinal diseases are nausea, vomiting, bloating, abdominal distention, diarrhea, constipation, and pain. For this reason, when functional disease is being considered as a cause of symptoms, it is important that the presence of non-functional diseases be excluded (ruled out). In fact, the exclusion of non-functional diseases usually is more important in evaluating patients who are suspected of having functional disease. This is so, in large part, because the tests for diagnosing functional disease are complex, not readily available, and often not very reliable.

Irritable bowel syndrome (IBS) is one of the most common ailments of the bowel (intestines) and affects an estimated 15% of persons in the US. The term, irritable bowel, is not a particularly good one since it implies that the bowel is responding irritably to normal stimuli, and this may or may not be the case. The several names for IBS, including spastic colon, spastic colitis, and mucous colitis, attest to the difficulty of getting a descriptive handle on the ailment. Moreover, each of the other names is itself as problematic as the term IBS.

IBS is best described as a functional disease. The concept of functional disease is particularly useful when discussing diseases of the gastrointestinal tract. The concept applies to the muscular organs of the gastrointestinal tract; the esophagus, stomach, small intestine, gallbladder, and colon. What is meant by the term, functional, is that either the muscles of the organs or the nerves that control the organs are not working normally, and, as a result, the organs do not function normally. The nerves that control the organs include not only the nerves that lie within the muscles of the organs but also the nerves of the spinal cord and brain.

Some gastrointestinal diseases can be seen and diagnosed with the naked eye, such as ulcers of the stomach. Thus, ulcers can be seen at surgery, on x-rays, and at endoscopies. Other diseases cannot be seen with the naked eye but can be seen and diagnosed with the microscope. For example, celiac disease and collagenous colitis are diagnosed by microscopic examination of biopsies of the small bowel and colon, respectively. In contrast, gastrointestinal functional diseases cannot be seen with the naked eye or with the microscope. In some instances, the abnormal function can be demonstrated by tests, for example, gastric emptying studies or antro-duodenal motility studies. However, these tests often are complex, are not widely available, and do not reliably detect the functional abnormalities. Accordingly, by default, functional gastrointestinal diseases are those involving the abnormal function of gastrointestinal organs in which abnormalities cannot be seen in the organs with either the naked eye or the microscope.

Occasionally, diseases that are thought to be functional are ultimately found to be associated with abnormalities that can be seen. Then, the disease moves out of the functional category. An example of this would be Helicobacter pylori infection of the stomach. Many patients with mild upper intestinal symptoms who were thought to have abnormal function of the stomach or intestines have been found to have an infection of the stomach with Helicobacter pylori. This infection can be diagnosed by seeing the bacterium and the inflammation (gastritis) it causes under the microscope . When the patients are treated with antibiotics, the Helicobacter, gastritis, and symptoms disappear. Thus, recognition of Helicobacter pylori infection removed some patients' diseases from the functional category.

The distinction between functional disease and non-functional disease may, in fact, be blurry. Thus, even functional diseases probably have associated biochemical or molecular abnormalities that ultimately will be able to be measured. For example, functional diseases of the stomach and intestines may be shown ultimately to be caused by reduced levels of normal chemicals within the gastrointestinal organs, the spinal cord, or the brain. Should a disease that is demonstrated to be due to a reduced chemical still be considered a functional disease? I think not. In this theoretical situation, we can't see the abnormality with the naked eye or the microscope, but we can measure it. If we can measure an associated or causative abnormality, the disease probably should no longer be considered functional.

Despite the shortcomings of the term, functional, the concept of a functional abnormality is useful for approaching many of the symptoms originating from the muscular organs of the gastrointestinal tract. This concept applies particularly to those symptoms for which there are no associated abnormalities that can be seen with the naked eye or the microscope.

While IBS is a major functional disease, it is important to mention a second major functional disease referred to as dyspepsia, or functional dyspepsia. The symptoms of dyspepsia are thought to originate from the upper gastrointestinal tract; the esophagus, stomach, and the first part of the small intestine. The symptoms include upper abdominal discomfort, bloating (the subjective sense of abdominal fullness without objective distension), or objective distension (swelling, or enlargement). The symptoms may or may not be related to meals. There may be nausea with or without vomiting and early satiety (a sense of fullness after eating only a small amount of food).

The study of functional disorders of the gastrointestinal tract often is categorized by the organ of involvement. Thus, there are functional disorders of the esophagus, stomach, small intestine, colon, and gallbladder. The amount of research on functional disorders has been focused mostly on the esophagus and stomach (such as dyspepsia), perhaps because these organs are easiest to reach and study. Research into functional disorders affecting the small intestine and colon .Digestrol™ alleviates abdominal stress that is makes you feel like a cage ball!

IBS and small intestinal bacterial overgrowth (SIBO)There is a striking similarity between the symptoms of IBS and a condition known as small intestinal bacterial overgrowth (SIBO).The entire gastrointestinal tract, including the small intestine, normally contains bacteria. The number of bacteria is greatest in the colon (at least 1,000,000,000 bacteria per ml of fluid) and much lower in the small intestine (less than 10,000 bacteria per ml of fluid). Moreover, the types of bacteria within the small intestine are different than the types of bacteria within the colon. SIBO refers to a condition in which abnormally large numbers of bacteria (at least 100,000 bacteria per ml of fluid) are present in the small intestine, and the types of bacteria in the small intestine resemble more the bacteria of the colon than the small intestine.

The symptoms of SIBO include excess gas, abdominal bloating and distension, diarrhea, and abdominal pain. A small number of patients with SIBO have chronic constipation rather than diarrhea. When the overgrowth is severe and prolonged, the bacteria may interfere with the digestion and/or absorption of food, and deficiencies of vitamins and minerals may develop. Loss of weight also may occur. The symptoms of SIBO tend to be chronic; a typical patient with SIBO can have symptoms that fluctuate in intensity over months, years, or even decades before the diagnosis is made.

It has been theorized that SIBO may be responsible for the symptoms in at least some patients with IBS. The estimates run as high as 50% of patients with IBS. Support for the SIBO theory of IBS comes from the observation that many patients with IBS are found to have an abnormal hydrogen breath test, a test used for diagnosing SIBO. In addition, some patients with IBS have improvement of their symptoms after treatment with antibiotics, the primary treatment for SIBO. Moreover, small, scientifically sound studies have shown that treatment with probiotics (“good” bacteria) improves the symptoms of IBS. Although there are several ways in which probiotics may be having their beneficial effect, one way is by affecting the existing bacteria in the small intestine. If this is indeed the mechanism of action, it would support the theory that SIBO is a cause of IBS. Nevertheless, it has not been determined if this is the mechanism of action of probiotics in IBS.

Although the theory that SIBO causes IBS is tantalizing and there is much anecdotal information that supports it, the rigorous scientific studies that are necessary to prove or disprove the theory have just begun. Nevertheless, many physicians have already begun to treat patients with IBS for SIBO. In addition, a lack of rigorous scientific studies demonstrating benefit from antibiotics and probiotics has not stopped physicians from using them for treating patients.

Treatment of IBS based on the theory of small intestinal bacterial overgrowth.

The two most common treatments for SIBO among patients with IBS are oral antibiotics and probiotics. Probiotics are live bacteria that when ingested by an individual, result in a health benefit to the individual. The most common probiotic bacteria are lactobacilli (also used in the production of yoghurt) and bifidobacteria, both of which are found in the intestine of normal individuals. There are numerous explanations for how probiotic bacteria might benefit individuals; however, the beneficial mechanism of action has not been identified clearly. It may be that the probiotic bacteria inhibit other bacteria in the intestine that may be causing symptoms, or it may be that the probiotic bacteria act on the host’s intestinal immune system to suppress inflammation.

Several antibiotics either alone or in combination are reported to be successful in treating SIBO in patients with IBS. Treatment success, when measured by either improvements in symptoms or by normalization of the hydrogen breath test, ranges from 40-70%. When one antibiotic fails, doctors may add another antibiotic or change to a different antibiotic, but the doses of antibiotic, the duration of treatment, and the need for maintenance treatment to prevent recurrence of SIBO have not been adequately studied. Most physicians use standard doses of antibiotics for one to two weeks. Probiotics may be used alone, in combination with antibiotics, or for prolonged maintenance. When probiotics are used, it probably is best to use one of the several probiotics that have been studied in medical trials and shown to have effects on the small intestine, (though not necessarily in SIBO). The commonly-sold probiotics in health-food stores may not be effective. Moreover, they often do not contain the stated bacteria or the bacteria are dead. Following are some options for treatment:

* neomycin orally for 10 days (One observation that has been made is that neomycin eradicates methane-producing bacteria and alleviates constipation.)
* neomycin orally for 10 days (One observation that has been made is that neomycin eradicates methane-producing bacteria and alleviates constipation.)
* metronidazole (Flagyl) for 7 days
* levofloxacin (Levaquin) combined with metronidazole (Flagyl) for 7 days
* rifaximin (Xifaxan) for 7 days. Rifaximin is a unique antibiotic that is not absorbed from the intestine, and, therefore, acts only within the intestine. Because very little rifaximin is absorbed into the body, it has few important side effects. Higher-than-normal doses of rifaximin (1200 mg/day for 7 days) were superior to standard lower doses (400 or 800 mg/day) in normalizing the hydrogen breath test in patients with SIBO and IBS; however, it is not yet known whether the larger dose is any better at suppressing symptoms.
* Commercially available probiotics such as VSL#3 or Flora-Q which are mixtures of several different bacterial species have been used for treating small intestinal bacterial overgrowth and IBS, but their effectiveness is not known. Bifidobacterium infantis 35624 is the only probiotic that has been demonstrated to be effective for treating patients with IBS.
Digestrol™ is the FINAL ANSWER for IBS and colitis sufferers that conquers constipation, cramps, and bulging eruptive contractions caused by irritated bowel syndrome. That’s why we made it condition specific, that’s what it’s all about!

The initial approach to IBS-treatment or testing--depends on the patient's symptoms and their duration. If the symptoms clearly fit the definition for IBS and have been present for years without change, then there is less need for extensive testing to exclude other intestinal and non-intestinal diseases. Rather, treatment that is directed at specific symptoms, as discussed previously, can begin. The role of antibiotics and/or probiotics is currently being studied.On the other hand, if the symptoms are of recent onset (such as weeks or months), progressively worsening, severe, or associated with "warning" signs, then early testing is appropriate. Warning signs include loss of weight, nighttime awakening, rectal bleeding, and signs of inflammation, such as fever or abdominal tenderness. Testing also is appropriate if, in addition to symptoms of IBS, there are other prominent symptoms that are not part of IBS (e.g., abdominal distention, increased flatus, or vomiting). Finally, testing is warranted if attempts at treating the symptoms of IBS are unsuccessful.
If there are symptoms that suggest non-IBS diseases, tests that are specific for these conditions should be done first. The reason is that if these other tests disclose disease other than IBS, it may not be necessary to do additional testing. Examples of symptoms and possible testing include:
* Vomiting: upper gastrointestinal endoscopies to diagnose inflammatory or obstructing diseases; and gastric emptying studies and/or electrogastrography to diagnose impaired emptying of the stomach.
* Abdominal distention with or without increased flatulence: upper gastrointestinal and small intestinal x-rays to diagnose obstructing diseases; and hydrogen breath testing to diagnose SIBO.
* Constipation without pain: colonoscopy or barium enema to exclude colonic cancer; marker studies to diagnose slow colonic transit; and ano-rectal motility studies to diagnose rectal muscle disorders
For a patient with typical symptoms of IBS who requires testing to exclude other diseases, the testing might reasonably include a standard screening panel of blood tests and stool specimens for examination for parasites, pus, and blood. A plain x-ray of the abdomen might be done during an episode of abdominal pain (to look for intestinal blockage or obstruction). Testing for lactose intolerance or a trial of a strict lactose-free diet should be done. Colonoscopy (and, possibly, esophago-gastro-duodenoscopy, or EGD) would be the next test, probably with multiple biopsies of the colon (and stomach and duodenum if EGD is done). Finally, small intestinal x-rays might be done.
For a patient with typical symptoms of IBS who requires testing to exclude other diseases, the testing might reasonably include a standard screening panel of blood tests and stool specimens for examination for parasites, pus, and blood. A plain x-ray of the abdomen might be done during an episode of abdominal pain (to look for intestinal blockage or obstruction). Testing for lactose intolerance or a trial of a strict lactose-free diet should be done. Colonoscopy (and, possibly, esophago-gastro-duodenoscopy, or EGD) would be the next test, probably with multiple biopsies of the colon (and stomach and duodenum if EGD is done). Finally, small intestinal x-rays might be done.
If all of the above appropriate testing reveals no disease that could be causing the symptoms, other tests should be considered. These tests include hydrogen breath testing to diagnose SIBO and antro-duodenal and colonic motility studies to diagnose intestinal muscle or nerve disorders. These studies probably should be done at centers that have experience and expertise in diagnosing and treating these diseases.
We even took it one step further and commissioned a clinical study on 22 Digestrol™ users. Patients were instructed to take Digestrol™, 2 tablets 2 times per day over a period of 3 months. All patients were assessed initially and every 2 weeks. Over the testing period, Digestrol™ showed a significant improvement in digestion and a definite relief of IBS symptoms of all patients.

Over time, such endotoxins put increasing amounts of stress on the body, and organisms such as bacteria, viruses and fungi will seize that opportunity to multiply and flourish. Their toxic waste may soon upset the balance of good and bad bacteria in the intestines, and as a result, organisms that normally inhabit the gastrointestinal tract in smaller numbers without causing harm (such as candida, a typically benign yeast) can proliferate. This can cause problems such as gas and bloating, constipation, abdominal pain and diarrhea.
Clinical research has proven that people who consume a diet high in fiber are less likely to suffer from constipation, irritable bowel syndrome, gastroesophageal reflux disease (GERD), and a host of other digestive disorders. That is because fiber helps to soak up toxins from the intestinal wall and sweep the colon free of debris. Found in fruits, vegetables, legumes, whole grains, nuts and seeds, fiber has also been shown to help prevent disease and support healthy weight management.
Increasing your intake of Essential Fatty Acids (EFAs) may also prove beneficial. These “good fats” include Omega-3 and Omega-6 fatty acids and are found primarily in cold-water fish and flax seeds. In addition to supporting the structure and function of every cell in the human body, EFAs help lubricate the colon and promote gastrointestinal health.
Research into probiotics, the beneficial bacteria that live in your intestines, has produced big breakthroughs in our understanding of health. Because studies have shown that probiotics interact with the immune system to prevent outbreaks of disease-causing organisms and keep the immune cells functioning at full capacity, adding an effective probiotic supplement to your daily diet may be the key to maintaining a healthy balance of bacteria in your intestines and improving your digestive health.Because enzymes are an essential part of all chemical processes that take place in the body, many people also benefit from taking a daily digestive enzyme. Although the best sources of enzymes are fresh fruits and vegetables, many Americans do not consume enough of these foods on a daily basis. When choosing an enzyme supplement, keep in mind that plant enzymes are best, as they are effective over a broader range of pH levels in the digestive system and thus provide the greatest benefit.
While it’s true that everything you put into your mouth will ultimately affect your digestive health, just a few simple changes in your diet can often provide significant benefits. Once you discover what works best for you and your body, you will soon be on your way to enjoying a healthier, happier lifestyle.